Different molecular mechanisms involved in spontaneous and oxidative stress-induced mitochondrial fragmentation in tripeptidyl peptidase-1 (TPP-1)-deficient fibroblasts

Keyphrases

• Mitochondrial Fragmentation 100%
• Neuronal Ceroid Lipofuscinosis 100%
• Mitochondrial Morphology 66%
• CLN2 66%
• Ceroid 33%
• Mitofusin 33%
• Impairement 33%
• Autophagic Pathway 33%
• Mitochondrial Oxidative Stress 33%
• Mitochondrial Fusion 33%
• Autosomal Recessive 33%
• Mitochondrial Network 33%
• Group of Eight 33%
• Late Infantile 33%
• Mitofusin 2 (Mfn2) 33%
• Form Cell 33%

INIS

• oxidation 100%
• fibroblasts 100%
• fragmentation 100%
• mitochondria 100%
• proteins 40%
• morphology 20%
• diseases 10%
• genes 10%
• rna 10%
• data 10%
• populations 10%
• inhibition 10%
• interference 10%
• pathogenesis 10%
• accumulation 10%
• pigments 10%
• antimycin 10%
• abundance 10%
• hydrolases 10%
• adenovirus 10%

Biochemistry, Genetics and Molecular Biology

• Batten Disease 66%
• Photosynthetic Pigment 33%
• Infantile Neuronal Ceroid Lipofuscinosis 33%